NUR 2349 / NUR2349: Professional Nursing I / PN 1 Exam 3 Review . Rasmussen

Document Content and Description Below

NUR 2349 Professional Nursing I PN1 Exam 3 1. The first leukocytes attracted to an injured tissue are the  Neutrophils 2. Fever that is seen in a client with an infectious disease i... s most likely caused by  3. Chronic inflammation can be caused by all of these  4. A nurse teaching strategy to reduce the development of antibiotic-resistant organisms is  5. A client with a known infection must be managed by using which identified method of precaution  6. All of the following are risk factors for delayed wound healing  7. In healing by primary intention, the wound fills in from  8. If a client has been taking a steroid drug wound healing will be delayed because the steroid drug  9. Rest and immobilization are important to wound healing because  10. Parents report their six-month-old daughter has diarrhea and vomiting for 24 hours which assessment finding do you expect to find that suggests fluid volume deficit  11. A priority assessment for client with fluid volume excess is  12. The cephalic vein runs along the  T 13. The ability to cause fluid movement across membranes  14. Which type of fluid can cause cells to swell and burst  15. Too much of which electrolyte can lead to respiratory depression and arrest  16. How does the thymus impact immune function?  17. What impact will thymectomy have on immune function?  18. What impact does a splenectomy have on immune function?  19. How are antibodies created  20. Primary immunodeficiency  21. Airborne transmission  22. Droplet transmission  23. Standard transmission  24. Vector transmission  25. Secondary immunity  26. Passive immunity  27. Active immunity  28. Two major problems that result from suppressed immune response  29. Medications that can cause immune suppression include  30. Suppressed immunity  31. Exaggerated immunity = hypersensitive response  32. Immunoglobulins  33. Five types of immunoglobulins Antibody  34. IgA  35. IgG  36. IgE  37. IgM  38. IgD  39. Immunosuppression  40. How does immunosuppression develop  41. Signs and symptoms of primary immunosuppression  42. Partial or complete suppression of immune response results in impaired ability to fight infection or disease  ... 43. Immune suppression is inherited at Birth or acquired through chemo, radiation, surgery, corticosteroids, HIV  ... 44. How does HIV cause immunosuppression?  45. Congestive heart failure  46. Inflammation signs and symptoms  47. Inflammation risk factors for development  48. Inflammation how is it treated  49. Inflammation physiology  50. Inflammation possible complications  51. Infection signs and symptoms  52. Infection risk factors for development  53. Infection how is it treated  54. Infection possible complications  55. Inflammatory response the nurse should monitor and assess  56. Exudate  57. Portal of exit  58. Mode of transmission  59. Portal of entry  60. Rheumatoid arthritis signs and symptoms  61. Rheumatoid arthritis complications  62. Rheumatoid arthritis treatment  63. Primary treatment for rheumatoid arthritis  64. Osteoarthritis most common medication used  65. Acquired immunity  66. Innate immunity  67. How to assess for cancer in a patient  68. Caution   69. Spleen  70. Lymph nodes  71. Thymus  72. Bone marrow  73. Hyponatremia  74. Hypernatremia  75. Therapeutic range for sodium  76. Therapeutic range for potassium  77. Hypokalemia  78. Hyperkalemia  79. Therapeutic range of calcium  80. Hypocalcemia  81. Hypercalcemia  82. Therapeutic range for magnesium  83. Hypo magnesia  84. Hyper magnesia  85. CBC with differential  86. Neutropenia  87. Neutrophils  88. Two classes of neutrophils  89. Shift to the right  90. Shift to the left  91. Basophils  92. Eosinophil  93. Monocytes  94. Lymphocyte  95. T lymphocytes  96. B lymphocytes  97. Dendritic cells  98. Mast cell  99. Chemotaxis  100. Primary intention  101. Secondary intention  102. Tertiary intention  103. Phases of wound healing  104. Inflammation phase  105. Proliferative phase  106. Remodeling phase  107. Therapeutic chloride range  108. Hypochloremia  109. Hypotonic  110. The correct order of the inflammation response  111. The classic signs swelling seen it in the employment Ori process results from  [Show More]

Last updated: 1 year ago

Preview 1 out of 18 pages